A decrease of both hypothalamic and cortical CCK occurred in the brain of rats killed 8 hours after hepatic artery ligation following portocaval anastomosis. Brain CCK depletion was not reproduced by exogenously provoked hyperammonemia nor by insulin-induced hypoglycemia, thus suggesting a central mechanism for the derangement of the CCKergic system in the course of acute liver failure.
Brain cholecystokinin depletion in rats with acute liver failure / C. Bonato, A. Malesci, R. Rosati, F. Vignati, M. Montorsi, M. Bersani, R. Abbiati, F. Salerno. - In: LIFE SCIENCES. - ISSN 0024-3205. - 44:2(1989), pp. 131-135.
Brain cholecystokinin depletion in rats with acute liver failure
A. MalesciSecondo
;R. Rosati;M. Montorsi;F. SalernoUltimo
1989
Abstract
A decrease of both hypothalamic and cortical CCK occurred in the brain of rats killed 8 hours after hepatic artery ligation following portocaval anastomosis. Brain CCK depletion was not reproduced by exogenously provoked hyperammonemia nor by insulin-induced hypoglycemia, thus suggesting a central mechanism for the derangement of the CCKergic system in the course of acute liver failure.
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