The present findings for familial Alzheimer's disease suggest a possible linkage to gene(s) on chromosome 21 for the early onset form and to chromosome 19 for the late onset. Since these results are not unequivocal, possible alternative hypotheses include the effect of genetic heterogeneity or of an oligogenic model of segregation for FAD.
A two-locus model for familial Alzheimer's disease? / F. Macciardi, M. C. Cavallini. - In: GENETIC EPIDEMIOLOGY. - ISSN 0741-0395. - 10:6(1993), pp. 437-41-441.
A two-locus model for familial Alzheimer's disease?
F. MacciardiPrimo
;
1993
Abstract
The present findings for familial Alzheimer's disease suggest a possible linkage to gene(s) on chromosome 21 for the early onset form and to chromosome 19 for the late onset. Since these results are not unequivocal, possible alternative hypotheses include the effect of genetic heterogeneity or of an oligogenic model of segregation for FAD.
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